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產(chǎn)品名稱：Anti-TUB 1抗體優(yōu)貨，TUB蛋白抗體

中文名稱：TUB蛋白抗體

英文名稱：Anti-TUB 1

產(chǎn)品編號(hào)：BYK-11536R

產(chǎn)品別名：F10B5.4; rd5; TUB 1; TUB; TUB_HUMAN; Tubby homologue; Tubby protein homolog 1; Tubby protein homolog.

本公司另供應(yīng)"Anti-TUB 1抗體,TUB蛋白抗體”的標(biāo)記有：Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647 AP APC Biotin Cy3 Cy5 Cy5.5 Cy7 FITC Gold HRP PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 RBITC.
產(chǎn)品規(guī)格：100ug/200ug
產(chǎn)品濃度： 1mg/1ml
抗體來(lái)源：兔源、鼠源、山羊
克 ?。?jiǎn)慰寺】贵w、多克隆抗體
抗體類(lèi)型：一抗
性 狀： 凍干粉或液體
保存條件：Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-TUB 1抗體優(yōu)貨，TUB蛋白抗體技術(shù)說(shuō)明，產(chǎn)品*，由于篇幅原因，更多信息請(qǐng)：
Anti-STAT6 /FITC  熒光素標(biāo)記信號(hào)轉(zhuǎn)導(dǎo)和轉(zhuǎn)錄激活因子6抗體IgG
Anti-STEAP1/FITC  熒光素標(biāo)記前列腺跨膜上皮抗原1抗體IgG
Anti-STK/CKⅠα/FITC  熒光素標(biāo)記屬絲/*蛋白質(zhì)激酶抗體IgG
Anti-STK/CKⅡα/FITC  熒光素標(biāo)記絲/*蛋白質(zhì)激酶抗體IgG
Anti-STK/CKⅡα/FITC  熒光素標(biāo)記屬絲/*蛋白質(zhì)激酶抗體IgG
Anti-Stra8/FITC  熒光素標(biāo)記*激活基因8抗體IgG
Anti-STRO-1/FITC  熒光素標(biāo)記STRO-1抗體IgG
Anti-SULT1E1/FITC  熒光素標(biāo)記雌激素硫酸轉(zhuǎn)移酶抗體IgG
Anti-SUMO/FITC  熒光素標(biāo)記類(lèi)泛素蛋白抗體IgG
Anti-Survivin/FITC  綠色熒光素標(biāo)記Survivin蛋白抗體IgG
Anti-SYK/FITC  熒光素標(biāo)記非受體型*蛋白激酶抗體IgG
Anti-SYT3/FITC  熒光素標(biāo)記突觸結(jié)合蛋白3抗體IgG
Anti-STAT3 /FITC  熒光素標(biāo)記信號(hào)轉(zhuǎn)導(dǎo)和轉(zhuǎn)錄激活因子3抗體IgG
Anti-STAT4 /FITC  熒光素標(biāo)記信號(hào)轉(zhuǎn)導(dǎo)和轉(zhuǎn)錄激活因子4抗體IgG
Anti-STAT5/FITC  熒光素標(biāo)記信號(hào)轉(zhuǎn)導(dǎo)和轉(zhuǎn)錄激活因子5（STAT5）抗體IgG
Anti-Socs 3/FITC 熒光素標(biāo)記細(xì)胞因子信號(hào)傳導(dǎo)抑制蛋白3抗體IgG
Anti-SOD1/FITC 熒光素標(biāo)記超氧化物歧化酶1抗體IgG

產(chǎn)品介紹：

In contrast to the rapid early-onset weight gain seen in ob/ob mice （1-3）, mutations in the tub gene lead to obesity gradually and strongly resemble late-onset obesity as seen in the human population （4）. In addition to excessive deposition of adipose tissue, mice with the tub phenotype also suffer retinal degeneration and neurosensory hearing loss （4-6）. The tripartite character of tubby phenotype is strikingly similar to human obesity syndromes such as Alstr鰉 （5） and Bardet-Biedl （6）. A candidate for the tub gene has been described （4）. A G芓 transversion in this candidate gene eliminates a donor splice site in the 3' coding region resulting in a larger transcript containing an unspliced intron （4）. A second prematurely truncated mRNA transcript with the unspliced intron was found to be expressed in the brains of tubby mice at a 2-3 fold higher rate as compared to B6 mice （4）. It has been postulated that the phenotypic features of tubby mice can be attributed to cellular apoptosis triggered by the expression of a mutated tub gene （4）.