上海博研生物工程研究中心為您現(xiàn)貨提供*的產(chǎn)品與服務，本中心產(chǎn)品齊全，保證實驗的有效性，提供，是您科研實驗產(chǎn)品的一站式平臺，咨詢。！

產(chǎn)品名稱：Anti-Huntingtin顧客好評,神經(jīng)性舞蹈病蛋白抗體
中文名稱：神經(jīng)性舞蹈病蛋白抗體
英文名稱：Anti-Huntingtin
產(chǎn)品編號：BYK-11699R
產(chǎn)品別名：HD; HD protein; HD_HUMAN; HDH; HTT; Huntingtin; HUNTINGTON CHOREA; Huntington disease protein; Huntington's disease protein homolog; IT 15; IT15; OTTMUSP00000026909; ZHD; AI256365; C430023I11Rik.

本公司另供應“神經(jīng)性舞蹈病蛋白抗體”的標記有：Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647 AP APC Biotin Cy3 Cy5 Cy5.5 Cy7 FITC Gold HRP PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 RBITC.
產(chǎn)品規(guī)格：100ug/200ug
產(chǎn)品濃度： 1mg/1ml
抗體來源：兔源、鼠源、山羊
克 ?。簡慰寺】贵w、多克隆抗體
抗體類型：一抗
性 狀： 凍干粉或液體
保存條件：Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-Huntingtin顧客好評,神經(jīng)性舞蹈病蛋白抗體技術(shù)說明，產(chǎn)品*，由于篇幅原因，更多信息請：

Anti-TNF-α /HRP  辣根過氧化物酶標記腫瘤壞死因子抗體IgG
Anti-CD34/Biotin  *標記兔抗人、大、小鼠、豬、兔、狗CD34抗體IgG
Anti-TOPOⅡα/DNA TPⅡα /FITC  熒光素標記DNA拓普西異構(gòu)酶Ⅱ抗體IgG
Anti-TNF-α /RBITC  紅色熒光素標記腫瘤壞死因子抗體
Anti-TP53/FITC  熒光素標記抗腫瘤P53抗體IgG
Anti-t-PA/FITC  熒光素標記組織型纖溶酶原激活劑抗體IgG
Anti-TPH /FITC  熒光素標記*羥化酶抗體IgG
Anti-TPO/FITC  熒光素標記兔抗甲狀腺過氧化物酶抗體IgG
Anti-TRA16/FITC  熒光素標記睪丸激素受體相關(guān)蛋白16抗體IgG
Anti-TRA16 /FITC  熒光素標記睪丸激素受體相關(guān)蛋白/孤兒素受體相關(guān)蛋白抗體IgG
Anti-TRADD/FITC  熒光素標記有死亡區(qū)的腫瘤壞死因子受體1相關(guān)蛋白抗體IgG
Anti-TPⅠ/FITC  熒光素標記拓普西異構(gòu)酶Ⅰ抗體IgG
Anti-TRAF6 /FITC  熒光素標記腫瘤壞死因子受體相關(guān)因子6抗體IgG
Anti-TP-1/TEP1/FITC  熒光素標記端粒酶相關(guān)蛋白1抗體IgG
Anti-TPⅡA/TOP2a/FITC  熒光素標記DNA拓普西異構(gòu)酶ⅡA抗體IgG
Anti-TRAIL/Apo2L /FITC  熒光素標記腫瘤壞死因子相關(guān)凋亡誘導配體/凋亡素2配體抗體IgG
Anti-Transthyretin /FITC  熒光素標記轉(zhuǎn)甲狀腺素蛋白抗體IgG
Anti-TRBF1 /FITC  熒光素標記端粒體復制結(jié)合因子1抗體IgG

產(chǎn)品介紹：
Huntingtin is a protein that contains a polyglutamine region. When the number of glutamine repeats exceeds 35, the gene encodes a version of Huntingtin that leads to Huntington’s disease （HD）. When the polyglutamine stretch is mutated, Huntingtin acts within the nucleus to induce neurodegeneration by a cell-specific apoptotic mechanism. Loss of Huntingtin activity is unlikely to be the cause of HD, and it has been proposed that the expanded glutamine repeat region may induce an abnormal interaction between the mutant protein and other cellular proteins. Huntingtin interacts with a variety of proteins including HAP1, glyceraldehyde phosphate dehydrogenase （GAPDH） and HIP1.