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產(chǎn)品名稱：Anti-Fx1A抗體,腎刷狀緣抗體直銷
中文名稱：腎刷狀緣抗體
英文名稱：Anti-Fx1A
產(chǎn)品編號：BYK-1338R
產(chǎn)品別名：gp330; RTEα5; LRP2_RAT; Low-density lipoprotein receptor-related protein 2; LRP-2; Glycoprotein 330; Megalin.

本公司另供應(yīng)“腎刷狀緣抗體”的標(biāo)記有：Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647 AP APC Biotin Cy3 Cy5 Cy5.5 Cy7 FITC Gold HRP PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 RBITC.
產(chǎn)品規(guī)格：100ug/200ug
產(chǎn)品濃度： 1mg/1ml
抗體來源：兔源、鼠源、山羊
克 ?。簡慰寺】贵w、多克隆抗體
抗體類型：一抗
性 狀： 凍干粉或液體
保存條件：Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-Fx1A抗體,腎刷狀緣抗體直銷技術(shù)說明，產(chǎn)品*，由于篇幅原因，更多信息請：

Anti-BADH2 （Betaine-aldehyde dehydrogenase ）  BADH2抗體
Anti-Bassoon/BSN（zinc finger protein 231）  鋅指蛋白231抗體
Anti-Batroxobin  蛇毒巴曲酶抗體
Anti-Batroxobin  蛇毒巴曲酶抗體
Anti-Bax （Bcl-2 Assaciated X protein）  Bax（小鼠來源抗體）
Anti-Bax（Bcl-2 Assaciated X protein）  Bax抗體
Anti-Bcl-2（B cell lymphoma/leukemia-2）  Bcl-2抗體
Anti-Bcl-xL  Bcl-xL蛋白抗體
Anti-BBC3/PUMA（bcl-2 binding component 3）  p53正向細(xì)胞凋亡調(diào)控因子抗體
Anei-ATX/Autotaxin/E-NPP2（Ectonucleotide pyrophosphatase/phosphodiesterase family member 2）  自分泌運(yùn)動因子抗體
Anti-Aurora B  極光激酶B抗體
Anti-B7-H1/PDL1/CD274（programmed death ligand 1）  程序性死亡配體1抗體
Anti-B7-DC/PDL2/CD273（programmed death ligand 2）  程序性死亡配體2抗體
Anti-ICOS-L/B7-H2/CD275（B7 homolog 2）  誘導(dǎo)協(xié)同刺激分子配體CD275抗體
Anti-B7H4 （ V-set domain containing T cell activation inhibitor 1 ）  B7H4抗體
Anti-BACE/ASP2  β分泌酶抗體
Anti-Bad （BCL-xL/BCL-2-associated death promoter）  相關(guān)死亡促進(jìn)因子Bad抗體
Anti-Bad （BCL-xL/BCL-2-associated death promoter）  相關(guān)死亡促進(jìn)因子Bad抗體
Anti-phospho-BAD（pSer128）  磷酸化相關(guān)死亡促進(jìn)因子抗體
Anti-Bak（BCL2 homologous antagonist/killer）  Bcl-2同源拮抗劑Bak抗體

產(chǎn)品介紹：

Binding of anti-Fx1A to Heymann nephritis antigens （HA） on rat glomerular epithelial cells （GECs） in culture leads to capping and disappearance of antigens from the cell surface. This process may contribute to the formation of glomerular subepithelial immune deposits in vivo. The authors differentially extracted GECs to determine whether HA redistribution is mediated by cytoskeletal components. Observations were made by phase-contrast and immunofluorescence microscopy on primary and passaged GECs in monolayer culture and by spectrofluorimetry on GECs in suspension.