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Anti-HCV-NS3抗體,丙型肝炎病毒-NS3抗體實實在在

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本公司提供高質量“Anti-HCV-NS3抗體,丙型肝炎病毒-NS3抗體實實在在",咨詢

上海博研生物工程研究中心為您現(xiàn)貨提供*的產(chǎn)品與服務,本中心產(chǎn)品齊全,保證實驗的有效性,提供,是您科研實驗產(chǎn)品的一站式平臺,咨詢。!

產(chǎn)品名稱:Anti-HCV-NS3抗體,丙型肝炎病毒-NS3抗體實實在在
中文名稱:丙型肝炎病毒-NS1抗體
英文名稱:Anti-HCV-NS3
產(chǎn)品編號:BYK-0219R
產(chǎn)品別名:Serine protease NS3; Hepacivirin; NS3P; POLG_HCVBK.

本公司另供應“丙型肝炎病毒-NS1抗體”的標記有:Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647 AP APC Biotin Cy3 Cy5 Cy5.5 Cy7 FITC Gold HRP PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 RBITC.
產(chǎn)品規(guī)格:100ug/200ug
產(chǎn)品濃度: 1mg/1ml
抗體來源:兔源、鼠源、山羊
克 隆:單克隆抗體、多克隆抗體
抗體類型:一抗
性 狀: 凍干粉或液體
保存條件:Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-HCV-NS3抗體,丙型肝炎病毒-NS3抗體實實在在技術說明,產(chǎn)品*,由于篇幅原因,更多信息請:

Anti-EphA2 R(Ephrin A2 Receptor)  兔抗*蛋白激酶A2受體抗體
Anti-EphA4 R(Ephrin A4 Receptor)  *蛋白激酶A4受體抗體
Anti-EphB2 R/Eph receptor B2 (Ephrin type B receptor 2)  Eph receptor B2 抗體
Anti-EPOR  紅細胞生成素受體抗體
Anti-EphB6R/Eph receptor B6(Ephrin type-B receptor 6)  *蛋白激酶受體B6抗體
Anti-ER-α(Estrpgen Receptor alpha)  雌激素受體α
Anti-ERAB  內質網(wǎng)Aβ相關結合蛋白抗體
Anti-ERCC1(DNA excision repair protein ERCC-1)  DNA切除修復蛋白1抗體
Anti-phospho-ERK1(pThr202/pTyr204)  抗磷酸化絲裂原活化蛋白激酶1抗體
Anti-ERK1/2(Mitogen-activated protein kinase 1/2)  絲裂原活化蛋白激酶1/2抗體
Anti-phospho-ERK1/pERK(pThr202/pThr204)+Anti-phospho ERK2(pThr183/pTyr185)  抗人、大、小鼠磷酸化絲裂原活化蛋白激酶1/2抗體
Anti-Eph receptor B2  Eph receptor B2抗體
Anti-MAPK1/ERK3 (Mitogen-activated protein kinase 3; P44MAPK)  絲裂原活化蛋白激酶3抗體
Anti-MAPK4(Mitogen-activated protein kinase 4)  絲裂原活化蛋白激酶4抗體

產(chǎn)品介紹:

Core protein packages viral RNA to form a viral nucleocapsid, and promotes virion budding. Modulates viral translation initiation by interacting with HCV IRES and 40S ribosomal subunit. Also regulates many host cellular functions such as signaling pathways and apoptosis. Prevents the establishment of cellular antiviral state by blocking the interferon-alpha/beta (IFN-alpha/beta) and IFN-gamma signaling pathways and by inducing human STAT1 degradation. Plays an important role in virus-mediated cell transformation leading to hepatocellular carcinomas. Interacts with, and activates STAT3 leading to cellular transformation. May repress the promoter of p53, and sequester CREB3 and SP110 isoform3/Sp110b in the cytoplasm. Also represses cell cycle negative regulating factor CDKN1A, thereby interrupting an important check point of normal cell cycle regulation. Targets transcription factors involved in the regulation of inflammatory responses and in the immune response: suppresses NK-kappaB activation, and activates AP-1. Mediates apoptotic pathways throught association with TNF-type receptors TNFRSF1A and LTBR, although its effect on death receptors-induced apoptosis remains controvertial. Enhances TRAIL mediated apoptosis, suggesting that it might play a role in mediated apoptosis, suggesting that it might play a role in immune-mediated liver cell injury. Secreted core protein is able to bind C1QR1 at the T-cell surface, resulting in down-regulation of T-lymphocytes proliferation. May transactivate human MYC, Rous sarcoma virus LTR, and SV40 promoters. May suppress the human FOS and HIV-1 LTR activity. May alter lipid metabolism by interacting with hepatocellular proteins involved in lipid accumulation and storage.

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