上海博研生物工程研究中心為您現(xiàn)貨提供*的產(chǎn)品與服務(wù)，本中心產(chǎn)品齊全，保證實驗的有效性，提供，是您科研實驗產(chǎn)品的一站式平臺，咨詢。！

產(chǎn)品名稱：Anti-HCV-NS4a抗體,丙型肝炎病毒-NS4a抗體實實在在
中文名稱：丙型肝炎病毒-NS4a抗體
英文名稱：Anti-HCV-NS4a
產(chǎn)品編號：BYK-0213R
產(chǎn)品別名：HCV-NS4a; Non-structural protein 4A; NS4A;POLG_HCVBK.

本公司另供應(yīng)“丙型肝炎病毒-NS4a抗體”的標(biāo)記有：Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647 AP APC Biotin Cy3 Cy5 Cy5.5 Cy7 FITC Gold HRP PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 RBITC.
產(chǎn)品規(guī)格：100ug/200ug
產(chǎn)品濃度： 1mg/1ml
抗體來源：兔源、鼠源、山羊
克 ?。簡慰寺】贵w、多克隆抗體
抗體類型：一抗
性 狀： 凍干粉或液體
保存條件：Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-HCV-NS4a抗體,丙型肝炎病毒-NS4a抗體實實在在技術(shù)說明，產(chǎn)品*，由于篇幅原因，更多信息請：

Anti-MKP-1/DUSP1（Mitogen activated protein kinase phosphatase 1）  絲裂原活化蛋白激酶磷酸酶-1抗體
Anti-MKP-2/DUSP4（Mitogen activated protein kinase phosphatase 2）  絲裂原活化蛋白激酶磷酸酶-2抗體
Anti-ER-α（Estrpgen Receptor alpha）  雌激素受體α抗體
Anti-ER/PR/c-erbB-2 Kit（mouse monoclonal）  ER/PR/c-erbB-2檢測試劑盒（鼠單抗）
Anti-phospho-ER-α（pSer167）human  抗人磷酸化雌激素受體α抗體
Anti-phospho-ER-α（pTyr537）mouse ret  抗人、大、小鼠、牛、犬磷酸化雌激素受體α抗體
Anti-phospho-ERK1（pThr202/pTyr204）  抗磷酸化絲裂原活化蛋白激酶1抗體
Anti-ERK1/2（Mitogen-activated protein kinase 1/2）  絲裂原活化蛋白激酶1/2抗體
Anti-phospho-ERK1/pERK（pThr202/pThr204）+Anti-phospho ERK2（pThr183/pTyr185）  抗人、大、小鼠磷酸化絲裂原活化蛋白激酶1/2抗體
Anti-Eph receptor B2  Eph receptor B2抗體
Anti-MAPK1/ERK3 （Mitogen-activated protein kinase 3; P44MAPK）  絲裂原活化蛋白激酶3抗體
Anti-MAPK4（Mitogen-activated protein kinase 4）  絲裂原活化蛋白激酶4抗體

產(chǎn)品介紹：

Function : Core protein packages viral RNA to form a viral nucleocapsid, and promotes virion budding. Modulates viral translation initiation by interacting with HCV IRES and 40S ribosomal subunit. Also regulates many host cellular functions such as signaling pathways and apoptosis. Prevents the establishment of cellular antiviral state by blocking the interferon-alpha/beta （IFN-alpha/beta） and IFN-gamma signaling pathways and by inducing human STAT1 degradation. Thought to play a role in virus-mediated cell transformation leading to hepatocellular carcinomas. Interacts with, and activates STAT3 leading to cellular transformation. May repress the promoter of p53, and sequester CREB3 and SP110 isoform 3/Sp110b in the cytoplasm. Also represses cell cycle negative regulating factor CDKN1A, thereby interrupting an important check point of normal cell cycle regulation. Targets transcription factors involved in the regulation of inflammatory responses and in the immune response: suppresses NK-kappaB activation, and activates AP-1. Could mediate apoptotic pathways through association with TNF-type receptors TNFRSF1A and LTBR, although its effect on death receptor-induced apoptosis remains controversial. Enhances TRAIL mediated apoptosis, suggesting that it might play a role in immune-mediated liver cell injury. Seric core protein is able to bind C1QR1 at the T-cell surface, resulting in down-regulation of T-lymphocytes proliferation. May transactivate human MYC, Rous sarcoma virus LTR, and SV40 promoters. May suppress the human FOS and HIV-1 LTR activity. Alters lipid metabolism by interacting with hepatocellular proteins involved in lipid accumulation and storage. Core protein induces up-regulation of FAS promoter activity, and thereby probably contributes to the increased triglyceride accumulation in hepatocytes （steatosis）.