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產(chǎn)品名稱：Anti-HIV1 gp160抗體,艾滋病病毒-1包膜糖蛋白抗體勁爆優(yōu)惠
中文名稱：艾滋病病毒-1包膜糖蛋白抗體
英文名稱：Anti-HIV1 gp160
產(chǎn)品編號：BYK-0785R
產(chǎn)品別名：HIV1 ENV （gp160）; HIV1 gp160; HIV1gp160; HIV-1 gp160; env; Env polyprotein; Envelope glycoprotein gp160; Glycoprotein 120; Glycoprotein 41; HIV1; HIV1 clade a; HIV1/Clade A; SU; Surface protein; ENV_HV1MV.

本公司另供應(yīng)“艾滋病病毒-1包膜糖蛋白抗體”的標記有：Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647 AP APC Biotin Cy3 Cy5 Cy5.5 Cy7 FITC Gold HRP PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 RBITC.
產(chǎn)品規(guī)格：100ug/200ug
產(chǎn)品濃度： 1mg/1ml
抗體來源：兔源、鼠源、山羊
克 ?。簡慰寺】贵w、多克隆抗體
抗體類型：一抗
性 狀： 凍干粉或液體
保存條件：Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-HIV1 gp160抗體,艾滋病病毒-1包膜糖蛋白抗體勁爆優(yōu)惠技術(shù)說明，產(chǎn)品*，由于篇幅原因，更多信息請：

anti-rGST（Recombinant Glutathione S-T Ahents）  重組*轉(zhuǎn)移酶GST標簽蛋白抗體
Anti-GSR/GRase（glutathione reductase）  *還原酶抗體
Anti-GTP-CH-1  三磷酸鳥苷環(huán)水解酶抗體
Anti-GTSE-1（G2 and S phase-expressed protein 1）  G2期、S期應(yīng)答相關(guān)蛋白1抗體
Anti-A/H5N1-M2（Avian influenza Matrix Protein-2）  A型禽流感病毒H5N1-M2蛋白抗體
Anti-A/H1N1-M2 Human（Avian influenza Matrix Protein-2 Human）  A型人流感病毒H1N1-M2蛋白抗體
Anti-GROα/GRO-1/MGSA/CXCL1 protein （growthregulated oncogene α）  生長調(diào)節(jié)致癌基因α/黑素瘤生zhang刺激因子α /生長調(diào)節(jié)致癌基因-1抗體
Anti-MIP2/GROβ/CXCL2   巨噬細胞炎癥蛋白2抗體
Anti-GRO gamma/CXCL3  生長調(diào)節(jié)致癌基因gamma/黑素瘤生zhang刺激因子3抗體
Anti-GRP94/gp96  葡萄糖調(diào)節(jié)蛋白94抗體
Anti-GSK-3α （Glycogen Synthase Kinase-3 alpha）  葡萄糖合成激酶-3α抗體
Anti-GSK-3β （Glycogen Synthase Kinase-3 beta）（CT）  葡萄糖合成激酶-3β抗體
Anti-GSK-3β （Glycogen Synthase Kinase-3 beta）（CT）  葡萄糖合成激酶-3β抗體
Anti-phospho-GSK-3β（pSer9）   磷酸化葡萄糖合成激酶-3β抗體
Anti-GSK-3β （Glycogen Synthase Kinase-3β）（NT）  糖原合酶激酶-3β抗體

產(chǎn)品介紹：

Function : The surface protein gp120 （SU） attaches the virus to the host lymphoid cell by binding to the primary receptor CD4. This interaction induces a structural rearrangement creating a high affinity binding site for a chemokine coreceptor like CXCR4 and/or CCR5. This peculiar 2 stage receptor-interaction strategy allows gp120 to maintain the highly conserved coreceptor-binding site in a cryptic conformation, protected from neutralizing antibodies. Since CD4 also displays a binding site for the disulfide-isomerase P4HB/PDI, a P4HB/PDI-CD4-CXCR4-gp120 complex may form. In that complex, P4HB/PDI could reach and reduce gp120 disulfide bonds, causing major conformational changes in gp120. TXN, another PDI family member could also be involved in disulfide rearrangements in Env during fusion. These changes are transmitted to the transmembrane protein gp41 and are thought to activate its fusogenic potential by unmasking its fusion peptide.